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Determination of the reduction–oxidation potential of the thioredoxin-like domains of protein disulfide-isomerase from the equilibrium with glutathione and thioredoxin

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Additionally, increasing studies have revealed the important mechanisms in the communication of mitochondria with other parts of cells (16), including cytochrome c release to induce cell death, 5′-adenosine monophosphate-activated protein kinase activation to control mitochondrial fission and fusion, ROS generation to activate transcription factors, and mtDNA release to activate immune responses (Figure 1)